Aging & sun damage — firming & photo-repair
Stimulate collagen and elastin production, lift sagging skin and repair the accumulated damage of sun exposure and time.
Aged and sun-damaged skin is losing collagen faster than it builds it, while years of UV exposure have injured the cells that keep skin firm. MYS-Exo exosomes work on both fronts — switching production back on in the fibroblast, inhibiting the enzymes that break collagen down, and repairing UV-induced cellular damage.
Aging & Sun Damage
Stimulate collagen and elastin production, lift sagging skin and repair the accumulated damage of sun exposure and time.
Skin feels firmer, superficial dry lines soften
Fine lines fade, facial contour gets lifted
Skin elasticity improves significantly, aging slows down
How to use: Use consistently for long-term results. Apply on face and neck; daily sunscreen is essential.
Targeting principle & mechanism
Core functional proteins: TGF-β1/3, bFGF, VEGF, HGF, elastin, procollagen, MMP-1 inhibitors, HSP70
Targets: Dermal fibroblasts, elastic-fibre cells, photo-aged / aging areas
- Activates the TGF-β/Smad pathway to continuously generate collagen, elastin and fibronectin, thickening the dermis and firming sagging.
- Inhibits MMP-1 collagenase, reducing collagen loss and slowing skin laxity.
- VEGF boosts dermal blood supply, improving dullness from late nights, dry lines and static fine lines.
- Antioxidant repair of UV-induced cellular DNA damage, slowing photo-aging.
- Rebuilds the dermal–epidermal junction (DEJ), softening nasolabial folds, eye-area fine lines and laxity creases.
How exosomes target the skin
Core functional proteins: CD9 / CD63 / CD81 tetraspanins, integrins, ICAM adhesion proteins, chemokine receptor CXCR4
Targets: Full-thickness damaged / problem skin areas and their target cells
- Targeted recognition: the exosome membrane carries a molecular “postal code” that binds matching ligands on cells in the problem area, actively grabbing the lesion while barely retaining on healthy skin.
- Transdermal delivery: the 30–150 nm nanoscale matches stratum-corneum gaps; with its phospholipid bilayer it enters cells intact via both passive penetration and active endocytosis, without being broken down.
- Intracellular release: once inside the cell the membrane fuses and ruptures, releasing functional proteins that directly regulate cell genes and signalling pathways — improving skin from the root.